Until recently, Terson’s syndrome (TS) was defined as the development of vitreous hemorrhage occurring in tandem with a subarachnoid hemorrhage (SAH). However, the definition has been expanded to include SAH, increased intracranial pressure (ICP), traumatic brain injury, and any form of intracerebral hemorrhage (1). More than 90% of vitreous hemorrhage cases are caused by either direct trauma to the head and orbit or by diabetes; less than 6% are caused by Terson’s (2). TS affects men and women equally and has been reported in infants as young as 7 months of age (3,4). This article will serve as a brief summary on the proposed pathophysiology, diagnosis, clinical implication, and management of TS.